摘要:
“恐音症”一词最先被美国神经学家帕维尔·贾斯特波夫(Pawel Jastreboff)及玛格丽特·贾斯特波夫(Margaret Jastreboff)于 2000 年提出,恐音症患者起因于自主神经系统和大脑边缘神经系统之间联系异常的情绪反应,目前科学家对引起恐音症的具体解剖学生理位置所知甚少,但很有可能这种异常存在于患者中枢神经系统的结构当中。本文研究发现,在声音源传导到脑组织的两条反射通路中,短通路(丘脑、杏仁核)与长通路(丘脑、扣带回、大脑各区域相应皮质),恐音症患者的过激情绪反应正是短通路优先于长通路而出现“短路现象”且异常过强的联系所致。恐音症也不同于听觉过敏,因它通常只针对某些特定声音才会产生反应,笔者团队发现恐音症通常也有合并症的状况(例如:Post-Orgasmic Illness Syndrome, POIS 综合症)[1]。本文从古今中外知名医典历史病例与当代流行病学的调查发现,恐音症早已不是如西方医学界所说的罕见现象。
Abstract: The term "misophonia" was first proposed by American neurologists Pawel Jastreboff and Margaret Jastreboff in 2000. People with misophonia result from emotional responses that are abnormally connected between the autonomic nervous system and the brain's limbic nervous system. Little is known about the specific anatomy and physiology that causes misophonia, but the abnormality likely resides in the structure of the patient's central nervous system. This study found that among the two reflex pathways that transmit the sound source to the brain tissue, the short pathway: thalamus, amygdala, and the long pathway: thalamus, cingulate gyrus, and the corresponding cortex in each area of the brain. The excessive emotional response of patients with misophonia is caused by the "short-circuit phenomenon" and the abnormally strong connection that the short pathway takes precedence over the long pathway. Misophonia is also different from hyperacusis in that it usually responds only to certain sounds. My team found that misophonia often has comorbid conditions, such as Post-Orgasmic Illness Syndrome. This paper finds that misophonia is no longer a rare phenomenon as the Western medical circles say, based on historical cases and contemporary epidemiological investigations of well-known medical texts at home and abroad.